Women and Nolvadex use

This remedy is somewhat different from others since it is not an anabolic/androgenic steroid. For male and female bodybuilders, however, it is a very useful and recommended compound which is confirmed by its widespread use and mostly positive results.

Nolvadex belongs to the group of sex hormones and is a so-called antiestrogen. The normal application of Nolvadex is in the treatment of certain forms of breast cancer in female patients. With Nolvadex it is possible to reverse an existing growth process of deceased tissue and prevent further growth. The growth of certain tissues is stimulated by the body’s own estrogen hormone. This is especially true for the breast glands in men and women since the body has a large number of estrogen receptors at these glands which can bond with the estrogens present in the blood. If the body’s own estrogen level is unusu-ally high an undesired growth of breast glands occurs. However, in healthy women and particularly in men this is not the case. Despite this, it is mostly male bodybuilders who use Nolvadex, and fewer women. At first sight this seems somewhat inconceivable but when taking a closer look, the reasons are clear. Bodybuilders who take Nolvadex also use anabolic steroids at the same time. Since most steroids aromatize more or less strongly, i.e. part of the substance is converted into estrogens, male bodybuilders can experience a significant elevation in the normally very low estrogen level. This can lead to feminization symptoms such as gynecomastia (growth of breast glands), increased fat deposits and higher water retention.

The antiestrogen Nolvadex works against this by blocking the estrogen receptors of the effected body tissue, thereby inhibiting a bonding of estrogens and receptor. It is, however, important to understand that Nolvadex does not prevent the aromatization but only acts as an estrogen antagonist. This means that it does not prevent testosterone and its synthetic derivatives (steroids) from converting into estrogens but only fights with them in a sort of “competition” for the estrogen receptors. This characteristic has the disadvantage that after the discontinuance of Nolvadex a “rebound effect” can occur which means that the suddenly freed estrogen receptors are now able to absorb the estrogen present in the blood. For this reason the combined intake of Proviron is suggested. Nolvadex is also useful during a diet since it helps in the burning of fat. Although Nolvadex has no direct fat burning effect its antiestrogenic effect contributes to keeping the estrogen level as low as possible. Nolvadex should especially be taken together with the strong an-drogenic steroids Dianabol and Anadrol 50, and the various testosterone compounds. Athletes who have a tendency to retain water and who have a mammary dysfunction should take Nolvadex as a prevention during every steroid intake. Since Nolvadex is very affective in most cases it is no wonder that several athletes can take Anadrol 50 and Dianabol until the day of a competition, and in combination with a diuretic still appear totally ripped in the limelight. Those who already have a low body fat content will achieve a visibly improved muscle hardness with Nolvadex.

Several bodybuilders like to use Nolvadex at the end of a steroid cycle since it increases the body’s own testosterone production. These can occur after the discontinuance of steroids when the androgen level in relationship to the estrogen concentration is too low and estrogen becomes the dominant hormone. A very rare but all the more serious problem of Nolvadex is that in some cases it does not lower the estrogen level but can increase it. Another disadvantage is that it can weaken the anabolic effect of some steroids. The reason is that Nolvadex, as we know, reduces the estrogen level. The fact is, however, that certain steroids especially the various testosterone compounds can only achieve their full effect if the estrogen level is sufficiently high. Those who are used to the intake of larger amounts of various steroids do not have to worry about this. Athletes however, who predominantly use mild steroids such as Primobolan, Winstrol, Oxandrolone, and Deca-Durabolin should carefully consider whether or not they should take Nolvadex since, due to the compound’s already moderate anabolic effect, an additional loss of effect could take place, leading to unsatisfying results.

A rarely observed but welcome characteristic of Nolvadex is that it has a direct influence on the hypothalamus and thus, by an in-creased release of gonadotropine, it stimulates the testosterone production in the testes. This does not result in a tremendous but still a measurable increase of the body’s own testosterone. This effect, however, is not sufficient to significantly increase the testosterone production reduced by anabolic/androgenic steroids.


The side effects of Nolvadex are usually low in dosages of up to 30 mg/day In rare cases nausea, vomiting, hot flashes, numbness, and blurred vision can occur. In women irregular menstrual cycles can occur which manifest themselves in weaker menstrual bleeding or even complete missing of a period. Women should also be careful not to get pregnant while taking Nolvadex. It is important for fe-male athletes that Nolvadex and the “pill” not be taken together since the antiestrogen Nolvadex and the estrogen-containing pill negatively counterfeit each other.


The normal daily dosage taken by athletes corresponds more or less to the dosage indications of the manufacturer and is 10-30 mg/day To prevent estrogenic side ef-fects normally 10 mg/day are sufficient, a dosage which also keeps low the risk of reducing the effect of simultaneously taken steroids. Often it is sufficient if the athlete begins this preventive intake of Nolvadex only three to four weeks after the intake of anabolics. Athletes who have tendencies toward gynecomastia, strong water retention, and increased fat deposits with steroids such as Dianabol, Testosterone, Anadrol 50, and Deca-Durabolin usually take 20-30 mg/day The combined application of Nolvadex 20-30 mg/day and Proviron 25-50 mg/day in these cases leads to excellent results. The same is true for athletes who are in competition, and for women. Women, however, should do without the intake of Proviron or at least reduce the dose to one 25 mg tablet per day. Unfortunately, in most cases, a very pronounced gynecomastia (“bitch tits”) cannot be reduced by taking Nolvadex so that often surgery is required, surgery which is not paid for by health insurance. First signs of a possible gynecomastia are light pain when touching the nipples. The tablets are usually taken 1-2 x daily, swallowed whole without chewing, with some liquid during meals.

Deca Profile. Deca-Durabolin helps joints.

Deca Durabolin is actually the brand name for Organons version of the compound Nandrolone Decanoate. Deca-Durabolin is a 19-Nor compound (some would say that it is the 19-nor compound), and as such, it shares basically the same characteristics with all of them. One thing unique to Deca Durabolin, above nearly all steroids, is the mystique it has had for the last quarter of a century. On a personal level, Ive included Deca Durabolin in cycles at doses ranging from 100mgs/week to 2,000mgs per week.

First of all, Deca Durabolin (and Nandrolone in general) doesnt produce many estrogenic or androgenic side effects. This is because Deca Durabolin has a very low rate of aromatization (conversion to estrogen via the aromatase enzyme), roughly equal to 20% the rate of testosterone.

Deca Durabolin stores water in connective tissue, thus alleviating joint pain. I have no idea what “storing water in the joints” means. I have no idea how to really quantify that statement, or where it started. However, in one study of postmenapusal women, Deca Durabolin impoved collagen synthesis, and in another study Deca Durabolin increased bone mineral content.

Both of these studies used VERY low doses, which were far too low to promote muscle growth. In my estimation, based on these 2 studies, an athlete attempting to use Deca Durabolin only for these two effects (increasing bone mineral content and collagen synthesis) should be using 100mgs of Deca Durabolin every week. Thats actually a higher dose than those two studies used successfully. Even at of this dose, in HIV+ patients who have experienced significant wasting, a 100mg/E2W (every 2 weeks) injection of Deca Durabolin resulted in a “significant increase in weight”. Id never recommend that low of a dose for an athlete, but its evidence of Deca Durabolin strong anabolic properties. Deca Durabolin is a very nice anabolic, causing nice (albeit slow) gains in quality muscle. This could be due to its moderately strong binding to the Androgen Receptor, or its many positive non-Androgen-Receptor mediated effects. One such effect is nitrogen retention, which is a major factor in muscle growth and lean mass gains; in one study, with low-doses (65 mg/week) and high-doses of Deca Durabolin (200 mg/week), both low-doses and high-doses resulted in significant nitrogen retention (33-52 g nitrogen/14 days, representing gains of 0.5 to 0.9 kg lean tissue/week), and body weight increased by 4.9 +/- 1.2 kg, including 3.1 +/- 0.5 kg lean body mass, and treadmill exercise performance (cardiovascular fitness) also improved. Need I say that the higher doses in this study produced more gains?

Deca Durabolin also has a very long active life. We can see from the chart below that a 100mg shot Deca Durabolin produced relatively active and stable plasma nandrolone levels until day almost 10, hence once a week shots are all thats necessary for stable levels of nandrolond Deca-Durabolinnoate (as a side note, the nandrolone phenylpropionate used in this study was active, and only experienced a severe drop off around day 5, shooting NPP every 4th day is the way to go). Youll also note that higher blood plasma levels of Nandrolone are found with Gluteal injections as opposed to Deltoid injections.

In another study of HIV+ men we can see that Deca Durabolin ( 200mgs on week 1, 400 on week 2 and 600mgs for weeks 3-12) caused NO negative side effects in total or LDL cholesterol, triglycerides, or insulin sensitivity and there was a reduction of HDL cholesterol (8-10 points) in both groups. Also, in most studies with HIV+ subjects, Deca Durabolin also improved immune function.

So what do we know so far about this compound? So far, we know that Deca-Durabolin is a very safe drug for long term use, will help with joint problems, could improve immune function, and is highly (!) anabolic, and not very androgenic.

Deca Durabolin Side Effects

Many members of Steroid.com also complain of water-retention with this drug, and again, Im inclined to agree. Letrozole seems to be a preferred choice to combat this, and its my favorite for this use. This water retention would seem to make Deca Durabolin more suitable for bulking rather than cutting, although it can be successfully used for either.

Now for the worst news: Deca Durabolin is a progestin (as are all nandrolones), unfortunately; it happens to stimulate the progesterone receptor 20% as well as progesterone itself, and this opens the door for many possible unwanted side effects (water retention, acne, etc…). It must be noted that most of those are rare, though. This also may be the major reason that Deca Durabolin is such a suppressive drug when it comes to your natural testosterone levels. We can see from the chart below that a simgle measly 100mg injection of Deca Durabolin caused a total (100%) reduction of natural testosterone levels, and it took roughly a month to return those testosterone levels to baseline! All from 100mgs of Deca Durabolin!

The moral of this story? Always use Testosterone with your Deca Durabolin! I suggest 200mgs, minimum, to avoid impotence and sexual dysfunction. For an anabolic effect from that Testosterone, I recommend at least double that, with an equal amount of Deca Durabolin (minimum). Id also recommend taking an anti-progesteronic drug with Deca Durabolin.

Deca Durabolin Cycle

So where are we? Well, Id be comfortable recommending Deca Durabolinfor use in a bulking cycle at up to 600mgs/week for an extended duration (12-16 weeks), or up to 400mgs/week in a cutting cycle (again, for 12-16 weeks), as long as something to combat water retention is present. Whichever purpose you decide to use Deca Durabolin for, you still need to include Testosterone in your cycle and have some anti-progesteronic drugs on hand just in case.

Post Cycle Therapy (PCT), though beyond the scope of this profile, needs to be commented on. Due to the highly suppressive nature of Deca Durabolin, I will speculate that testosterone in a Deca Durabolin-inclusive cycle needs to be run for at least 2 additional weeks upon cessation of Deca Durabolin. We remember from the chart above that baseline testosterone levels took roughly a month to return. Hence, a nice long estered testosterone should be run about 2 weeks longer than Deca Durabolin, to prevent having a lag in time when the Deca Durabolin is not producing an anabolic effect, yet is still suppressing your natural testosterone levels. Id also suggest that a particularly aggressive PCT be run after your cycle; Nolvadex, HCG, and perhaps Clomid should all be utilized in an effort to restore your natural hormone levels as quickly and efficiently as possible.

Turn Up The Heat For Muscle Mass!

According to a new study that shows that heating muscle may increase muscle mass through the expression of heat shock proteins. The following research may persuade bodybuilders to use the sauna and hot tubs more often for muscle recuperation and muscle growth. Researchers reported that the muscles of animals exposed to heat stress stimulated increases in heat shock proteins, which not only caused the activation of satellite cells (skeletal muscle stem cells are responsible for the repair and hypertrophy of skeletal muscle), but also increased protein synthesis during the regeneration of injured skeletal muscle. It was strongly suggested by the researchers that the application of heat to injured or damaged muscle may facilitate recovery and enhanced satellite cell activation leading to enhanced muscle mass.

Want more? Also researchers reported that intermittent heat application (30 minutes on alternating days) to the muscles of rats enhanced muscle hypertrophy. You might want to go steal grandma’s heating pad after reading these research papers! Heat shock proteins have previously been shown to increase muscle protein synthesis and increase muscle mass. In fact, a single bout of heat stress has been demonstrated to increase muscle mass and protein synthesis. Heat shock proteins are stimulated by many factors, but originally were discovered to be increased by the application of heat, hence “heat shock proteins.”

Heat shock proteins (HSPs) are a group of proteins whose expression is increased when the cells are exposed to elevated temperatures or other stress. The exercise-related stressors that have been shown to induce HSPs are: elevated temperature, decreased glucose availability, increased intracellular calcium levels, increased catecholamines and reduced blood flow to muscle. Consequently, HSPs are also referred to as “stress proteins” and their upregulation is sometimes described more generally as part of the stress response because they are increased in response to numerous stressors. HSPs provide cellular protection and work together with the antioxidant system to prevent further damage to cells. Thompson28 observed a large HSP response to high-force eccentric exercise, but a reduced HSP response following a repeated bout of high-force eccentric exercise. Thus, the increases in HSPs after high-force eccentric exercise may have reduced further damage to muscle.

HSPs have recently been shown to be important for increasing muscle protein synthesis and muscle hypertrophy. Scientists believe that heat stress-associated muscle hypertrophy may be induced by calcium-related intracellular signals. It is speculated that the application of heat stress to muscles may activate the intracellular signals, which stimulates the protein synthesis and/or modifies the protein breakdown. Therefore, heat stress may modify the intracellular Ca2+ level in skeletal muscles. Others have reported that Ca+ signaling is important for mediating muscle hypertrophy. For example, Oishi et al. recently reported that muscle hypertrophy induced by muscle overload was associated with an increased intracellular calcium level and that treatment with a drug that blocks calcium signaling reduced muscle hypertrophy during muscle overload. Activation of calcium may be one of the intracellular signals for skeletal muscle hypertrophy in response to IGF and increased loading. Preheating of skeletal muscles is being experimented on as a means of preventing muscular atrophy during bed rest inactivity and also the possibility of exposing astronauts to heat stress to reduce anti-gravity-related muscle atrophy.

Back in 1995, researchers were examining how reduced blood flow or so-called tourniquets could increase muscle hypertrophy. They performed surgical procedures on rats to reduce blood flow to their legs; surprisingly, the rats’ legs grew in size due to reduced blood flow. Muscle fibers were 34 % larger from the reduced blood flow in the experimental group. The content of HSPs also increased in muscle, which the researchers speculated may be important for muscle hypertrophy. Additionally showed that overload of muscle caused muscle hypertrophy in rats and increased both myosin heavy-chain protein (a consequence of muscle cell remodeling) and HSP expression in the hypertrophied leg muscle compared to the control leg. Taken together, these data demonstrate a relationship between protein synthesis, muscle cell remodeling and HSP expression. HSPs are not only increased by heat and exercise, but also can be increased pharmacologically. Clenebuterol has previously been shown to cause an increase in HSPs in muscle, which may be a molecular pathway in which clenebuterol increases protein synthesis and increases muscle mass.

In a recent study, researchers were looking specifically at how the expression of HSPs affect muscle protein content and muscle differentiation (process in which precursor muscle cells become mature muscle fibers. It’s a necessary process for muscle growth). Both processes are important for increasing muscle mass. The researchers examined heat stress, GGA or heat stress plus GGA. The researchers found that the administration of the anti-ulcer drug, GGA, as well as the heat stress, increased the expression of HSPs and protein content in muscle cells. It was suggested that GGA may activate intracellular growth signals, which stimulates muscle growth and protein synthesis. The researchers concluded that the administration of GGA could be one of the useful tools to gain muscular mass not only in athletes, but also in patients during rehabilitation. Why, all of a sudden, do all bodybuilders feel a burning sensation in their chest?

If you are in the gym busting your ass, and your legs are on fire from the high intensity, then that burning sensation is more than determination— but rather HSPs being expressed! The degree of HSPs expressed in muscle seems to be influenced by the total amount of work performed in the gym. Researchers investigated the HSP response in trained humans exposed to four weeks of rowing exercise by taking serial muscle biopsies after each week of training. The expression of HSPs increased in relation with increasing amounts of exercise, and they suggested that the HSP response was related to the total amount of exercise. The same research group then revised this statement and found that more importantly than just total work volume, HSPs are also stimulated to a greater extent during high-intensity exercise. For example, elite male rowers had muscle biopsies performed during two parts of their training season and post-season. During the training season, the sessions were divided so that the volume was the same, but one group performed at a low intensity and the other group performed at a high intensity. At the end of the study, there were the largest increases in HSPs being expressed during the high-intensity training season while HSP levels declined during the post-season when training volume and intensity were reduced. A recent study also found that HSP was correlated with the concentration of blood lactate levels during exercise, which may be the reason for higher HSP expression during high-intensity exercise.

HSPs seem to be increased with exercise sessions that cause muscle damage. Protocols that are higher in intensity and performed eccentrically elicit significant increases in HSPs. A recent study showed that a single bout of maximal eccentric exercise increased HSPs by 203 % within 23 hours of exercise. Interestingly, heating muscle and intense exercise causes a greater rise in HSPs than either one alone. One study looked at two forms of eccentric training and HSP expression. Researchers compared high-force eccentric barbell curls and moderate aerobic-intensity downhill running for 30 minutes. HSPs increased during high-force barbell curls but not during aerobic downhill running. The study demonstrates that a high-force eccentric stimulus promotes HSP responses that are not seen following a moderate, mixed-contraction exercise modality such as downhill running. The researchers concluded that the lower training intensity used in downhill running failed to increase HSP levels because of insufficient training intensity. Additionally, it has been suggested that the eccentric overload causes an increase in HSPs, which play an important role in the regulation of muscle mass. Performing eccentric contractions seems to be a better stimulus of HSPs than concentric contractions. Eccentric contractions are known to promote more hypertrophy than concentric contractions. Although the mechanisms of why eccentric contractions increase muscle hypertrophy is not clear, it is known that the process of hypertrophy involves long-term changes in protein synthesis, and several suggests that increases of HSPs are involved in these processes. Thus, HSPs are stimulated by high-intensity training. High-intensity exercise can increase testosterone levels as well; it should be of no surprise that when testosterone is administered, there is an increase in HSP expression in fast-twitch muscle fibers.Remember that fast-twitch muscle fibers have the most potential for muscle growth! Testosterone increases muscle mass, so could testosterone be increasing protein synthesis by activation of HSPs?

For many years, heat treatment has been used as a therapy for muscle injuries. There are several different heat modalities, such as heat packs, whirlpools, ultrasound and recently, microwave hyperemia. Molecular mechanism responsible for muscle hypertrophy in response to various stimuli, such as strength training and bodybuilding, is still unclear. Recently, the application of heat stress has been found to induce hypertrophy in muscle cells and facilitate the recovery of muscle atrophied following muscle unloading. Although we generally don’t think of a sauna as anabolic, it could possibly be for bodybuilders. Heat stress has been shown to reduce muscle atrophy and increase muscle protein synthesis. For example, Naito et al. reported that heat stress before an eight-day period of hindlimb unloading (unloading causes rapid muscle atrophy) in rats reduced muscle atrophy. Compared with sedentary cage control rats, hindlimb unloading resulted in a 25 % atrophy and a 40 % decrease in the level of HSPs. When muscle unloading was pretreated with heat stress, muscle atrophy was only 17 % and had >20 % higher HSP levels than control after eight days of unloading. It was postulated that the elevated levels of HSPs could have retarded muscle atrophy by both maintaining protein synthesis and decreasing the rate of protein breakdown. Other researchers have reported similar findings; it was reported that if animals underwent hindlimb unloading and then were immediately exposed to heat stress, thereafter the muscle exposed to heat stress had accelerated protein synthesis and muscle mass as compared to animals that had nothing.

A direct role of HSPs in protection of skeletal muscle has been demonstrated in both animals and humans. An increase in HSPs may also be able to increase strength the day after a heavy workout. For example, animals that overexpress HSPs by 20 times greater than normal had a 63 % lower rate of strength decline as compared to normal animals three days after a session of lengthening contractions. High-intensity resistance training increases HSP in muscles of both older and younger individuals— although the average relative increase of HSP expression appears to be larger in the young. The HSP response to exercise is lower in older rats with more hypertrophy-prone fast-twitch muscle fibers, which may play a part in why older people don’t have the same increase in muscle mass as younger people. Others have demonstrated the failure of successful recovery of skeletal muscles of older animals is due to a blunted activation of HSPs following muscle damage and suggests that abnormal activation of HSPs may play a major role in the defective regeneration seen in muscles of old mice. The defect in HSPs may provide strong evidence for the protective role of HSPs in preventing damage and strength loss in skeletal muscle during the aging process.

It is known that exercise and heat stress induce muscle fiber injury or protein breakdown. Because HSPs function to maintain cellular function under stressful conditions, it is speculated that HSPs are involved with the repair of injured skeletal muscles exposed to stress. The exposure to heat, muscle damage by eccentric exercise and high-exercise intensity are potent stimulators of HSPs. HSPs are currently being examined as a possible anabolic treatment to athletes, as the British Journal of Sports Medicine researchers found that microwave hyperthermia to muscle increased HSPs in the legs of subjects. Who knows? Maybe you can get big by lying out in the sun and getting a tan! As far as supplements, glutamine may also enhance HSP expression. Researchers examined the role of glutamine in muscle under two environments: heat stress and resting. Glutamine has a stimulatory effect on the rate of protein synthesis in skeletal muscles subjected to heat stress, while it has no effect on the rate of protein synthesis in normal muscle fibers. Glutamine increases HSP expression only in heat-stressed muscle fibers. Therefore, HSPs may play a role in the mechanism by which glutamine enhances the rate of protein synthesis in heat-stressed muscle fibers. One final bit of wisdom— stay away from alcohol if you want to increase HSPs, as alcohol consumption blunts HSP expression in muscle.

Key Points:

  • Heat stress activates satellite cell activity, increases muscle mass and enhances muscle rejuvenation.
  • HSPs provide cellular protection and protects against further damage.
  • HSPs are increased in response to training intensity.
  • Eccentric exercise is a potent stimulator of HSPs.

Injectables Guide For Ladies

Ladies that have tried at least one mild oral steroid and now wish to add more mass to their bodies.

The chances of contracting side effect, and potential virilization, are higher with injectables than with anavar. Anavar is the mildest steroid for ladies without a doubt…too bad it is not in an injectable form.

Most common sides seen with the ladies are as follows and in no particular order. Some negatives can also be positives as you will note below

NEGATIVE SIDES - Ance, oily skin, clitoral enlargement beyond minimal, aggression, hair loss..male style, hair growth especially on upper lip and chin, hair loss, darkened hair growth, quickened hair growth on legs and arms, lowering of voice tone, distruption of normal menstral cycle, aggression.

MOST COMMON NEGATIVE SIDES - Oily skin, some hair growth, a little acne, some alteration in normal menstral cycle, minimal voice tone lowering, darkening body hair

PRECURSER TO VOICE LOWERING - If you get a squeaky voice, hoarse throat or voice, scratchy thoat, raspy throat or voice, a cough, or any ache in the throat then stop the steroid immediately as these are common warning signs of voice alterations

PERMANENT NEGATIVE SIDES - Voice lowering may improve immediately after a cycle A LITTLE but will NEVER return to normal. Some girls are affected minimally and still sound like a women and others end up sounding like a man if they continue with the steroid. Clit growth remains…androgenic swelling goes away but the GROWTH remains. Some long time and heavy users have grown a little penis(TRUE!)

POSITIVE SIDES - Feelings of well being, increased energy, decreased recovery time, aggression, heightened sex drive with small amount of clit enlargement, Muscle gain, strength gain, some reports of decreases in estrogenic fat ie”upper legs, butt, upper arms, abdomen.

PERMANENT POSITIVE SIDES - Mass and strength will largely remain IF you train and eat properly post cycle. Better sex, due to clit growth, but sex DRIVE returns to normal except for the possible increase in sex drive as a result of heightened enjoyment.

Pure anabolics vs androgenic anabolics.

There are only two injectables that are nearly pure anabolics and they are Nandrolone and primobolan. They are somewhat androgenic but one could classify them as anabolics. The more androgenic the hormone the greater are your chances for sides including virilization, but the greater your chances are for great muscle growth. The most androgenic hormones are Test and Tren and then perhaps winny, although some would disagree with me on the winny.

PRIMOBOLAN - Used by many ladies as it, along with nandrolone, is probably the least androgenic roid. CAREFUL….Primobolan is in the long acting ester enenthate and it clears slowly. Many ladies have experienced very bad sides from this roid due to the fact that they take too much per week (above 50-75mg). Injectable Primobolan is not our drug of choice because it is long acting and if sides come on that you don’t like then you have to ride them out until the roid clears your system and by the time it does permamnent sides may have set in. Daeo’s wife had a bad experience with Primobolan. If you start low you shouldn’t have too much trouble, if any, IMO as it is quite mild androgenically. Just remember though that it will not give quick muscle gains at all. The gains seen are usually quality muscle built at a slow steady rate. If more and quicker gains are needed then I would recommend Nandrolone.

ESTERS AND DOSE SCHEDULES. - An ester is a compound made up of hydrogen and carbon atoms. They are added to pure steroids or testosterone in order to slow the release of the active agent. Some esters have a short chain of hydrogen and carbon and others have longer chains. The longer the chain the longer the time release of the steroid. Propionate is a short ester and Decanoate is a long chain ester. 50mg of Nandrolne in a Phenylprop ester has more actual steroid than 50mg of Nandrolone in the Decanoate ester as some of the 50mg weight is taken up by the longer ester. THE DIFFERENCE IS MINIMAL! Never let anyone tell you that one type of steroid is more powerful than another type of the same steroid. These are myths . There is a myth that Testosterone Cypionate is more powerful than Testosterone Enanthate and this is a myth. In actuality the enanthate ester has one less carbon atom and 50 mg of the test will have a tiny amount more test than 50mg of cyp.


Most conservative…..Inject 50 mg of steroid or testosterone once and then let it completely clear the system. ie: Testosterone Propionate 50mg then wait a week before doing another injection. OR…Testosterone Cypionate 50mg and then wait for two weeks before injecting again. This system will give you some results and is very safe in regard to sides.

The next method is recommended by Bill Llewellyn and others. In this system one injects a dose and then lets it partially clear the system. It is thought that this clearance will not allow androgens to build up over time which of course can cause sides and virilization. This sytem will give better results and liitle sides. Examples are 25- 50mg Testosterone Propionate every 5-7 days or 50mg of Testosterone Cypionate every 10 days.

The last system will give best results. This system is favored by many and requires injecting AT LEAST as frequently as a male.
In this system you try to inject at least twice per half life. This keeps hormone spikes to a minimum. These more even blood levels result in better gains. There is also some evidence to suggest that spiking hormone levels cause more sides. Here is an example of this system…Testosterone Propionate 15mg every second day or 20 mg every third day. Also Testosterone Cypionate 20-25 mg twice per week or every forth day at most. Simply put the longer you are “on” the greater are your chances for virilization. An 8 week cycle is a good start. latter cycles can be longer once you get a feel for the hormone. A long low dose cycle of 12 weeks or more can be great.


Testosterone 25-50mg
Primobolan 50-75mg
Winistrol 50-70 mg 7-10mg per day INJECT DAILY for best results
Nandrolone 25-60mg
EQ 25-60mg

These doses will give you great gains with little chance of serious sides. If you are top level competitor then obviously these doses are quite moderate. I would not recommend any women go much above these doses unless they are darn serious about being competitive!


We do not recommend that you use more than one hormone at a time unless your are a top competitor. If you do stack your voice will almost certainly lower and other sides will be worse. If you MUST stack than we would recommend using a small dose of anavar with the injectable at perhaps 10mg per day, 5mg in the am and 5 mg in the pm.

While trying to increase mass while on injectables you must increase your protein intake to about 2 grams per pound of body weight. If you do not do this then your gains will be likely be less . I recommend a good low fat whey protein shake betwee three regular meals. Some people seem to get great gains with less but it is safer to go higher. Caloric intake also needs to go and the increased protein intake will help.
You can get good strength gains ,and some muscle gain, while on anavar, and not eat that much in the way of protein or calories, but this is not the case when trying to build some serious muscle.

The anabolic/catabolic diet

First, lets look at the ABCDE diet. Originally ABCDE Diet (Anabolic Burst Cycling of Diet and Exercise) was proposed by Torbjorn Akerfeldt in an old issue of Muscle Media 2000. Basically, you ate your heart out for two weeks, which would elicit all sorts of different anabolic responses and cause you to add muscle. Then for the next two weeks, you would turn things around by going on a strict Low-Caloric diet. Done correctly, you’d lose the fat you gained during your hog fest and keep most of the muscle. That’s of course in “theory” meaning it works like crap with the average bodybuilder. ABCDE diet is great for people who do not easily gain fat or who easily lose fat. But what if you rapidly gain fat and cannot seem to be able to get rid of it? What will happen is that by feasting for 2 weeks you will accumulate a tremendous amount of fat.

This accumulation occurs in two forms:

Each fat cell will hypertrophy. Once fat cells reach critical size they release certain growth factors which, in turn, build up new fat cells. This hyperplasia will occur in a matter of days if your adipocytes are already big enough, i.e; as you are fat in the first place. Of course, as the diet state, you can diet down the hypertrophy, but you cannot get rid of newly formed fat cells. As you repeat the ABCDE cycles you will add new fat cells. Intense training (because of cortisol) plus a high carb and high fat diet is a great way to induce adipose tissue hyperplasia in people who are prone to gain fat.

Your body fat percentage is the reflection of the size of the fat cells multiplied by their number. You can shrink fact cell size but only up to a certain point. You’re not able to lose fat cells. In other words, as you start a low calorie diet, you will only have a single variable to play with (reducing fat cell size). But once you have reduced fat cell size up to a point (which is quickly reached) they won’t shrink anymore. So you will not have any variables left.

The diet stops working and you are stuck at a high bodyfat percentage. The only way to lose fat cells is to use weird drugs, or to undergo liposuction. I think the concept of the ABCDE diet is valid but not appropriate for fat people or drug users. For those people I propose a variation of this diet that I have been using for some time. I call it the anabolic/catbolic diet (let’s call it ACD diet).

It is based on the fact that there is a constant turnover of protein in our muscles. We are constantly adding protein (anabolism) but also losing protein (catabolism). It is the balance between the two which determines the size of our muscles.

So whenever someone complains of only gaining 5 pounds a year, this is not strictly true. He gained maybe 200 pounds of muscle but he also lost 195 pounds. This (contrived) example points out that we are able to gain a tremendous amount of muscle every year. As we cannot hold on to those 100 pounds, it means we are doing something really wrong. As its name implies, the ACD diet is composed of two distinct phases.

An anabolic phase lasting 10 to 20 days and a catabolic phase lasting 5 to 10 days. When I say a catabolic phase, I really mean it; we do whatever it takes in order to lose muscle. Our body will detect something is very wrong and will attempt to stop this rapid muscle loss. It will deactivate the ATP-dependent proteolytic pathways. So far, almost no drug that I know of can do this. The more catabolism you seek, the more your body will fight it. After the catabolic phase, we will be in a position where both anabolism and catabolism are very low. But anabolism is ready to be accelerated to catch up growth while catabolism is reduced to a minimum for a while no matter what. During the anabolic phase, we will try to boost anabolism as much as possible.

During the first week, catabolism will stay very low. So, there will not be an anabolic catch up, there will be an overshoot. How do we do that? During the catabolic phase, the goal is to lose as much fat as possible no matter what the cost is for the muscle mass. I mean a low carbohydrate, low fat and low protein diet. If you are using creatine stop it. Oral anabolics will be stopped 48 hours before the beginning of this phase while 5 days before the injectables are stopped. Thyroid medication (T3) will be started at day one. Clenbuterol will be used at day one and maybe on day four or fine at massive doses (15 to 20 tabs). Of course, if you are new to Clenbuterol do not use that much. At that dose, used acutely you should feel sore all over which will only enhance catabolism.

Overtraining is a must and don’t forget about the daily (at least) one hour of aerobics. During your first few catabolic cycles, please do not play with so many variables. Avoid too much aerobics and don’t be too hard on the diet. Make it last only 5 days. As you get used to it, add days and variables. The following anabolic phase will start with either a carb load or a fat load lasting only on and a half days. But be careful about not eating too much. Many people get sick whenever they try to load on food after starving themselves. On the other hand, one or two days sick in bed is a good way to end up the catabolic phase but only if you are very advanced. The fat load consists of the following: on the last day of the catabolic phase do 2 hours of low intensity cardio. I like the rowing machine since it involves both lower and upper body. Repeat this cardio workout again on an empty stomach before the fat load.

As its name implies the fat load consists of eating fat with some proteins but no carbs at all. This will direct the fat inside the aerobically trained muscles not in the subcutaneous adipocytes. Whenever you are eating carbs along with fat, this fat will tend to be esterified in the subcutaneous adipose tissue not inside the muscles. I know the ABCDE diet tells otherwise, but oh well¦. Even though you are not eating carbs, don’t forget the creatine. It will find its way in the muscles without insulin. The oral anabolics will be started ruing the first day while the injectables will be used as late as possible the day before. Along with the carb up, use creatine and ALA. In the first few days, eat a high carb, low fat and moderate protein diet. The protein should be of the highest quality possible to make up for the moderate amount. The more protein eaten the sooner the ATP-dependent proteolytic pathway will recover.

Of course the thyroid medication, the Clenbuterol, the aerobics and the overtraining are stopped. In fact, it is better to reduce training to a bare minimum in the first few days. During the remainder of this phase, do not eat too much. Unless you want to gain fat cells, you should just eat over maintenance and no more. I do not think overfeeding builds up muscle. My belief is that the measuring techniques are biased.

Whenever you eat too much and gain weight, they are going to tell you have gained muscle mass along with the fat. But whenever you diet it down, the bias will take place the other way around. The apparatus will tell you that you have lost muscle along with the fat. Your overfeeding gains just evaporated.

The length of the anabolic phase is determined by the length and the severity of the catabolic phase. The shorter the latter is, the shorter the former should last. Of course, this kind of radical cycling is much more appropriate for steroid users than for drug free bodybuilders. It allows anabolics to keep working for a long time as it is frequently stopped during a short catabolic phase. This will fully restore their potency. In case you are a drug free bodybuilder, it is better not to go to the extreme. Shoot for a moderate catabolism not an extreme one.

Nandrolone PhenylPropionate by Accordo RX - Explained

Nandrolone Phenylpropionate was never really all that popular simply because of availability issues. Many of the pharmacy grade Nandrolone Phenylpropionate products range between 25mg-50mg/ml and are extremely expensive.

Nandrolone Phenylpropionate vs Decanoate.

Let’s calculate the amount accumulated in the body after 6 weeks of 500mg/deca. Let’s say you inject it once a week and we’ll give it a 1.5-week half-life. Note that injection frequency makes a huge difference in blood concentration stability but no difference in amount of esterified in the system

E (greek letter “sigma”) 500*e^(ln(1/2)n/1.5) from n=0 to n=6. So after 6 weeks, about 1300mg of esterified nandrolone remain in the body.

Now lets see how long, after the initial injection, it takes to reduce to a small enough amount that permits recovery.

1300*e^(ln(1/2)n/1.5) After 3 weeks, 325 mg of esterified remaiт after 6 weeks, 81 mg of esterified remain After 8 weeks, 32mg of esterified remain.

Nandrolone Decanoate is a long acting depot; it takes quite a while for it to “kick in” and clear out of the system. Depending on how much is used; it will take at least 4-6 weeks after the last shot for Deca to clear out. It also takes about 4 weeks for active blood levels to stabilize. This can easily add up to 8-10 weeks of “dead time” i.e. periods of time when blood levels are not consistent. These numbers apply to reasonable use of Nandrolone Decanoate; between 200-400mg a week. The more you use, the worst it gets. So a 10-week cycle of Deca can easily end up been a 16-week cycle when you account for clearance time (active blood levels). The first 4 weeks are also somewhat of a waste of time.

So that 10-week cycle ends up been 16 weeks; 6 weeks of optimal blood levels and 10 weeks of dead time. Not a very effective way to cycle.

With Nandrolone Phenylpropionate, you can bypass all that dead time. 19-Nortestosterone based drugs are known to shutdown HPTA very easily - think Trenbolone. Most bodybuilders will use Tren for around 6 weeks at the beginning of a cycle. Nandrolone Phenylpropionate should be used in a similar manner.

Here’s an example of a balanced cycle consisting of Nandrolone Phenylpropionate

Week 1-6: Dianabolbol
Week 1-6: Nandrolone Phenylpropionate
Week 1-8: Test Prop

It is a good idea to run Test 2 weeks past the Nandrolone Phenylpropionate, however; Nandrolone Phenylpropionate can be used as a stand-alone. Earlier, I compared Nandrolone Phenylpropionate to Tren. They are similar in some ways but Tren is much more androgenic and stronger in general.

Nandrolone Phenylpropionate shares some of the same sides associated with Deca (they are after all the same base compound). It should be noted that most of the sides that come with Deca are a result of its long ester. Decanoate ester is very hard to control and Nandrolone side effects are not easily countered like Testosterone related sides (Tamoxifen, anastrozole, finasteride…) Overall, Nandrolone is a milder compound than Testosterone and is better mg for mg (but that’s a matter of opinion)

Nandrolone Phenylpropionate should be injected at least every 3 days. A typical dose is 350mg-700mg a week for 5-8 weeks. It stacks very well with Winstrol, Dbol, Test, EQ, Anavar It does not stack well with Tren and especially Anadrol

Here are some good cycle suggestions:

Fast Acting Classic Test/Deca/Dbol cycle:

Week 1-6: Dianabol 30mg ED
Week 1-6: Nandrolone Phenylpropionate150mg EOD
Week 1-8: Test Prop 150mg EOD

Highly Anabolic cycles

Week 1-6: Nandrolone Phenylpropionate: 200mg E3D
Week 1-8: Anavar: 30mg ED

Week 1-6: Nandrolone Phenylpropionate: 200mg E3D
Week 1-8: Winny: 50mg ED

A good First cycle:

Week 1-6: Nandrolone Phenylpropionate: 150mg E3D
Week 1-4: Dbol: 25mg ED
(Week5-8: Anavar: 30mg ED - optional)

Nandrolone Phenylpropionate in a typical cycle

Week 1-10: EQ 400mg a week
Week 1-9: Test Cyp 600mg a week
Week 1-8: Nandrolone Phenylpropionate 200mg E3D
Week 10-13: Test Prop 150mg EOD

Nandrolone got a very bad rap with many bodybuilders; there is no reason to use Nandrolone Decanoate if Nandrolone Phenylpropionate available aside from year-round juicer using it for joint pain. Nandrolone is a tremendous bodybuilding drug that can take your physique to a whole different level but many people shy away from it because of what they have heard or experienced with Deca.

When Can I Start Taking Steroids

The problem with this is that most young bodybuilders can’t grasp the concept of the future and how long the road ahead really is even with the use of steroids. How do you convince a 14 year old that it will take years before he can look like the Pro bodybuilders in the magazines, and that he may never look like that even with all the drugs available in the world.

At what age should you be, before you consider using anabolic steroids? This question is not as easily answered as you may think it is. You cannot randomly just pick an age and say that this is the point at which you can now start to consider using anabolic steroids.

Between the ages of 12 and 26 a male’s hormone levels are on a steady rise until the age of 26. This is when these levels slowly start to decline until they are almost nonexistent by the ripe old age of 40.

When puberty starts in males at the age of 12 there is a huge flux in hormonal patterns in the body, which cause the growth of male characteristics, (deepening of the voice, growth of body hair, growth in height, etc.). These hormone levels increase by themselves so much that they can be compared to that of a mild anabolic steroid cycle. Therefore trying to add to what the body is doing on its own by adding in exogenous (outside) anabolic steroids is very counter productive.

Whenever any extra amount of anabolic steroids is added to the body, the body recognizes this extra level through a feedback loop in the human body known as the hypothalamus. Once the hypothalamus recognizes the increase in hormones which happens usually between 14 and 21 days, the body will shut off its own production of hormones until these levels decrease, along with increasing hormones to decrease these extra levels in the body (cortisone, estrogen). Cortisone and estrogen are 2 hormones in the body that bodybuilders do not need any extra. The easiest way to try to explain this without getting to complicated, is that the more anabolic steroids you put in your body, the more your body will try to lower those levels. When this happens bodybuilders get all the side effects that are normally associated with anabolic steroids use, gynecomastia (growth of fatty tissue underneath the breasts in males), hair loss, kidney damage, liver damage, and high blood pressure, just to name a few).

Before you consider the use of anabolic steroids you should have already reached your genetic potential. What is your genetic potential? To figure this out you should first look at the weight, height and build of other members in your family. Is this exact, no, but it is somewhere to start. If every male on both sides of your family is approximately 5’7” and weighs between 150lbs and 170lbs and they are all bald by the age of 25, then it would be a good guess that you will also fall somewhere in those ranges by the time you stop growing. Now with working out and eating correctly for 4 years lets say, you would be able to put on 15 or more pounds of muscle tissue (that would mean you now weigh 165-185 lbs.). This is what your genetic potential would be. Now if you started to use steroids at that point, 165-185 lbs you may be able to put on another 15-20 lbs (180-200lbs). If you had started using when you were 125 lbs., and gained 25lbs through the use of anabolic steroids, you would still be well short of what you could have gotten naturally (150lbs as compared to 180-200lbs), and now it will be much harder to try to gain another 30-40lbs.

So for a starting point lets say that you need to be at least 18 years of age before you consider using anabolic steroids. Now that we have a starting point, lets look at a few other factors that should be considered. Steroids DO NOT IN ANY WAY, SHAPE, OR FORM makes up for a good diet and workout program. Most people who use anabolic steroids feel that this is the time where they can be a little more relaxed in their workouts and diet. Actually this is when they should be even more strict. So before you can think of using at the age of 18, you will need 3 more solid years of good training and eating habits. Minor changes in diet and workouts can result in great gains in mass and muscle as well as strength.

No matter how much assistance you get from anabolic steroids, without proper nutrition and workouts you will be lucky to have any gains at all, let alone keep them after the cycle is over. The goal of using any substance, legal or not should be that after you stop using it you don’t loose all of that which you have fought to get. What would be the point of spending all that money (steroids are not free) to gain that 30lbs when you will loose it after the cycle is over anyway?

So the better question to ask instead of when can I take anabolic steroids, should be, what can I do to get all that I can out of my body without needing anabolic steroids?. In order to calculate my progress, I need to talk with my family and doctors, before I try to make a choice like that.

We will start with the age range of 14-16; this is when your hormones are raging. Your body is in full swing of making the best steroids that you can ever get, and it does all this without you even needing to do a single thing. At this point you should start with a solid exercise plan and a basic supplement plan in addition to the regular food that you need to be eating on a regular basis.

For workouts focus on the basic compound movements (Bench Press, Squats, Deadlifts, Barbell Curls, etc.) Working out 4 days a week with at least 8 hours of sleep a night is a great start. Add to that the extra protein that you should be taking and you will definitely start seeing a difference in your body.

By the age of 16-18 you will have had most of your growth spurts and you will be ready to change a few more things in your overall plan. You workouts can become a little more specialized as you start using different exercises. 

Somewhere between 18 and 21 you should be just about done growing, so what should you do differently now? Add more protein! You should be getting at the very least, your body weight in grams of protein per day! And that?s at the very least! By now you will have been working out consistently for quite a while and should know your body very well. What will work and what won?t work should be old news. There isn’t really anything new to add to what is already a great program, other than Tribulus and maybe a pre-workout supplement such as Ultimate Orange. After this point, you can start to consider the use of steroids. What about them? Are they as terrible as everyone seems to think they are? As long as they are used correctly, I don?t think so. When considering their use, I feel that orals should be used as late as possible. These are most harmful on the body and therefore should not be used for a very long time.

Another thing to consider, other than the side effects I have already spoken of, is your sex drive. Some anabolic steroids will make your sex drive almost nonexistent and will have a big effect on your sperm count. These drugs in particular should try to be avoided. That leaves mild anabolics. Although they are the safest to use, they are expensive, illegal, and require the use of a needle. Which most first time users do not want to use.

I told you that this is not something that should be passed off very easily; you shouldn?t have to make the mistakes that most of us make by using steroids to early in your life. If used correctly, I think they are fine, but look at what it takes in order to use them correctly. Have you been working out for 5 years straight without more the 2 weeks off every 6 weeks? Do you eat every 2-3 hours, 6 times a day without missing a meal? Do you get 8 hours of sleep every night? This is something that can have a huge effect on your body for the rest of your life, so don?t make that choice in 10 minutes. Good luck and keep growing.

Deca Durabolin vs Equipoise

Deca Durabolin, also known as Deca and Nandrolone Decanoate, is an anabolic androgenic steroid that is used by bodybuilders to bulk up. While this steroid is second to none for stimulating dramatic improvements in terms of recuperation time between workouts, it also has the ability to promote nitrogen retention and protein synthesis. Not only this, Deca Durabolin is easy on the liver and unarguably one of the best performance enhancing drugs for promoting strength, muscle size, and wet gains.

Deca Durabolin is characterized by a very low rate of aromatization that makes it ideal even for athletes who are prone to estrogenic side effects, though progesterone sides (some prolactin) are usual. Moreover, they can expect slow but steady gains in terms of muscle mass, strength, and muscle tissue. Deca Durabolin is more suited to longer steroid cycles and is generally stacked with Testosterone Suspension, Testosterone Enanthate , Testosterone Propionate, Testosterone Cypionate, Anadrol, Dianabol, and Sustanon 250. Most athletes prefer stacking Deca Durabolin and Dianabol as they share a synergistic effect with each other.

Equipoise has come a long way from just being a veterinary drug to transform itself into one of the most popular and recommended bodybuilding drugs. This extremely anabolic and moderately androgenic steroid stimulates appetite and has the unmatched ability of increasing endurance. A perfect pre-contest drug, Equipoise can dramatically increase Hemoglobin and Hematocrit (the number of red blood cells and the percentage of red blood cells) that in turn promote greater “pumps” during intense workouts. One of the biggest advantages associated with Equipoise is that this anabolic androgenic steroid can stimulates the release of erythropoietin in the kidneys. A big majority of athletes prefer stacking Equipoise with Testosterone Enanthate, Testosterone Cypionate, Winstrol, Anadrol, Sustanon 250, and Testosterone Propionate.

Considered by a big majority of bodybuilders as a dream steroid, Deca Durabolin is superior to Equipoise. This is primarily because Deca Durabolin is better than Equipoise in terms of retention of muscle mass and body strength gains. However, Deca Durabolin users should always follow a very strict post cycle therapy with drugs such as Arimidex, Clomid , or Nolvadex to protect against excess estrogen. There is one issue with Deca Durabolin that’s a big downside, Deca Durabolin stays in the body for a long duration (approximately 2 years) and is therefore not the ideal drug for athletes who are tested regularly.

Equipoise, on the other hand, is preferred by experienced athletes and bodybuilders who can be satisfied with moderate gains that are sustaining in nature. While Equipoise is associated with androgenic side effects like hair loss, growth of body and facial hair, and acne, Deca Durabolin is often associated with gynecomastia and fluid retention. It is widely believed that Deca Durabolin is more suited for bulking cycles while Equipoise is more suitable for cutting cycles. The use of Equipoise is associated with slow but steady gains in terms of muscle mass and strength with very few estrogenic side effects. Equipoise can be a part of longer cycles. The fact that Equipoise promotes red blood cell production and stimulates appetite slightly puts it ahead of Deca Durabolin.

Deca-Durabolin Nandrolone Decanoate use, dose , effects and risks

Nandrolone is by far one of the most popular anabolic steroids available. This is due to the compound’s affinity for being highly anabolic but relatively mild in terms of androgenic side effects. Nandrolone is the base steroid 19Nor-testosterone, meaning that it is like testosterone in appearance except for the absence of a carbon atom in the 19th position. This small change makes a major difference in the characteristics of the compound. Notably, this change makes nandrolone a less potent agonist of the androgen receptor. This of course reduces the chance that a user will experience androgenic side effects. Instead of forming dihydrotestosterone when encountering the 5 alpha reductase enzyme like testosterone does, nandrolone will form dihydronandrolone. Dihydronandrolone is extremely mild in terms of it’s antagonizing the androgen receptor. Therefore androgenic side effects should be far less likely to occur with nandrolone than with testosterone.

It is of note however that nandrolone is believed to have some activity as a progestin in the body. Although progesterone is a c-19 steroid, removal of this group as in 19-norprogesterone creates a hormone with greater binding affinity for its corresponding receptor. The compound will stimulate the progesterone receptor, along with progesterone. Side effects associated with this activity are similar to some of those related to estrogenic ones including water retention, acne, and gynecomastia among others. However these side effects are fairly rare in comparison to more androgenic compounds.

Now, with the reduced chance of negative side effects associated with the use of nandrolone one would assume that the gains in lean body mass that a user could expect to make would also be reduced. This is not true however. Nandrolone is 2.4 times as anabolic as testosterone when compared gram for gram. This does not mean that a user will gain 2.4 times as much muscle if using nandrolone instead of testosterone however. It is not that easy. It does show how powerful this drug is and how it can help a user make serious gains in muscle mass.


Due to the active life of the compound, users would only need to administer nandrolone decanoate once per week to maintain fairly stable of the drug. However, since the vast majority of users will stack nandrolone with testosterone and the most commonly used esters need to be injected more frequently most will simply injected it when it is convenient, such as twice a week with their testosterone enanthate or cypionate.

Anecdotally most users report that the full benefits of nandrolone decanoate will not be felt until the fifth, sixth or even seventh week of using the compound. This necessitates that users run the drug for at least eleven or twelve weeks for a cycle of the compound to have the results that a user expects. Of course, since nandrolone decanoate is a relatively mild drug many users have run it for extremely lengthy cycles with little in the way of serious side effects.

Most inexperienced male users will start using about 400mgs of nandrolone decanoate during their first cycles of the compound. Like most drugs users have used quite large doses of the compound, with diminishing returns being seen as these doses go higher and higher.

Females also have found that nandrolone can be quite effective while offering relatively mild side effects. Doses ranging from about 50 to 150mgs per week seem to be the norm for most first time users of the compound, again with these doses increasing as users try to achieve more and more with the compound.

Side Effects/Risks

Estrogenic effects are not a major concern with use of nandrolone due to it’s low rate of aromatization. However it can cause progesterone-like effects in some users. Commonly reported sides effects associated with nandrolone are such things as acne/oily skin, insomnia, diarrhea, and nausea. These of course are coupled with the common side effects most often associated with anabolic steroids including testicular atrophy, gynecomastia (including lactation in some cases), and sexual dysfunction.

To combat sexual dysfunction most users will stack testosterone with nandrolone. The obvious choice to be used with nandrolone decanoate is testosterone enanthate or cypionate due to the length of the esters being similar. How much testosterone one would need to take to ward off side effect associated with use of nandrolone and lack of natural testosterone production varies from individual to individual. Some have anecdotally reported that a low dose similar to 200mgs per week is enough. Others state that they need to run several hundreds more milligrams per week of testosterone than nandrolone to combat the effects. There is a small minority of individuals that also report having no sexual dysfunction from the drug even while running it without any type of testosterone. This variance again demonstrates that individuals will react to a compound differently than others.

This effect also points to the fact that nandrolone is extremely suppressive to a user’s natural testosterone production. Even at relatively small doses, much smaller than those that would be used by those wanting to reap the anabolic effects that the compound can offer, nandrolone can suppress the natural production of testosterone of a user so much that it can take up to thirty days after the drug cycle is complete for it to fully recover. For this reason it is imperative that a user run a well planned post cycle therapy after using nandrolone.

Nandrolone is relatively safe in terms of a user’s lipid profile and cholesterol. In some studies it has even been shown to actually improve HDL cholesterol levels. A major increase in a user’s blood pressure or their liver toxicity should not be noticed with this compound either. Nandrolone is relatively mild in these respects. Since Nandrolone is a progestinic anabolic steroid. Some special precautions need to be taken to ensure that side effects do not get out of control as a result of a rise in prolactin levels. Using compounds such as bromocriptine, cabergoline and/or vitamin b6 have all been shown and reported to help lower prolactin levels. The drug Femara (letrozole) is also effective for use with nandrolone as it will regulate the progesterone and estrogen receptors in the body, therefore preventing some of the negative side effects associated with the compound.

A word of caution also for those that may plan on stacking nandrolone with trenbolone. Trenbolone is a strong progestin, much stronger than even nandrolone. By running these two compounds concurrently the user will suffer from extremely high levels of prolactin. This in turn will force the user to pay special attention to progesterone-like side effects and using compounds to prevent them, as well as having to run a particularly aggressive post-cycle therapy due to severe suppression of the hypothalamus pituitary testicular axis. Some users have anecdotally reported that they have suffered no ill effects of running the two compounds together, but it is a definite risk. One must weigh the costs versus the benefits.

How to Create The Perfect Steroid Cycle Stack for Mass, Cutting and PCT

So you want to create the perfect cycle for yourself. So how do you go about this? Well there’s a lot of things you need to know before you can sit down and create yourself a
perfect cycle.

The most important thing you need to know is what your exact goals are for this cycle. From here you can figure out what steroids are right for you and at what dosages.

What about steroids, ancillaries, and other drugs do you need to know? You need to know the basics of the most popular drugs available.


  • Testosterone (Enanthate, Cypionate, Propionate)
  • Deca-Durabolin
  • Equipose
  • Dianabol
  • Winstrol
  • Anadrol
  • Anavar
  • Trenbolone
  • Primobolan


  • Nolvadex/ (Tamoxifen) 
  • Arimidex/ (Anastrozole)
  • Clomid 
  • HCG 
  • Proviron (technically a steroid, but oft considered an ancillary) 
  • Finasteride

Other Bobyduilding/Performance Enhancing Drugs:

  • Clenbuterol
  • Cytomel

There are of course many other types of steroids, ancillaries and sports enhancing drugs but they are extremely rare. We won’t go into a full discussion about each of the drugs above, but will just list properties of the drugs and state which steroids have those properties.

-Large Mass Steroids: Testosterone, Deca-Durabolin, Anadrol, Dianabol and to a lesser extent: Equipose, Primobolan
-Strength Steroids: Testosterone, Dianabol, Trenbolone and to a lesser extent: Anavar
-Steroids that have low/no aromatization: Equipose, Primobolan, Anavar, Trenbolone, Winstrol
-Steroids that raise red blood cell count: Equipose, Anadrol and to a lesser extent: most others
-Low-Lean Mass Steroids: Winistrol, Anavar, Trenbolone
-Steroids with direct fat-burning properties: Testosterone, Trenbolone, Anavar
-Mostly Anabolic Steroids: Deca-Durabolin, Equipose, Primobolan, Winstrol, Anavar
-Highly Anabolic Androgens: Dianabol, Anadrol, Trenbolone
-Mostly even Androgenic/Anabolic Steroids: Testosterone
-Steroid most likely to cause aggression: Trenbolone
-Liver Toxic Steroids: Dianaboll, Winstrol, Anadrol, Anavar
-Short Acting Steroids: Testosterone Propionate, Dianabol, Winstrol, Anadrol, Anavar, Trenbolone
-Long Acting Steroids: Testosterone Enanthate, Testosterone Cypionate, Deca-Durabolin, Equipose, Primobolan,
-Progestins: Deca-Durabolin, Anadrol
-Prolactins: Trenbolone
-Acts like an estrogen: Anadrol
-Anti-Progestin: Winistrol
-Anti-Aromatases: Arimidex, Aromasin, Proviron
-Anti-Estrogens: Nolvadex, Clomid
-Anti-Androgens: Finasteride
-Fat Burners: Clenbuterol, Cytomel
-Stimulates LH release: HCG
-Aids HPTA recovery: Clomid, Nolva

Ok so now that you know what drugs do what, we can begin to discuss what properties a cycle should have. From there we can begin to see how these drugs can be combined to form a “stack.?The idea behind the stack is to create a synergy between the drugs involved to give an effect that’s greater than the sum of the parts.

Mass Cycles:
These are cycles where all out mass is required. Here we give no consideration to fat gain, water gain or any of that. We are just looking to pack on as much muscle as possible (don’t forget, water and fat are GOOD for muscle gains).

To get all out mass, we need to attack our system from all angles. We need anabolic steroids that are highly androgenic and highly anabolic. We need anabolic steroids that are known to pack on a lot of mass. In general, anabolic steroids that do not aromatize, do not activate the AR and do not pack on a lot of mass aren’t needed. For injectables we would rather have long acting esters than short ones, as the long acting esters tend to pool up in your blood and generally leave you with more hormone at any given point. For orals we prefer those that either aromatize heavily, or cause an explosion of mass by similar estrogenic properties. The use of orals is mainly to kick off the mass cycle, gives you near instant results and puts your body in a good anabolic state when the long acting esters kick in.

With all that said the best steroids for mass are: Testosterone Enanthate, Testosterone Cypionate, Deca Durabolin, Dianabol and Anadrol.

Cutting Cycles:
Realize that with the exception of Testosterone, Trenbolone, and Anavar, no steroid has a direct impact on fat burning. Even Testosterone, Trenbolone and Anavar have limited effects on fat burning. You shouldn’t go into a cutting cycle with the mindset of “These steroids are going to help me lose fat,” Instead you should think of the steroids as muscle sparring. Basically you’re using them to preserve the muscle that you have, while, diet, cardio and your true fat burners (like Clen, DNP and T3) work on the fat. All steroids listed above meet the first requirement; they will all help you retain muscle in a calorie deficient diet. However, if you are cutting you certainly do not want your steroids to be in the way either. Some steroids (drol) actually make it harder to loose fat. Others can bloat you up so bad that even with a low body fat percentage, most of your definition can be lost. So what we need here is steroids that are more androgenic than anabolic. We need steroids that have direct fat burning properties and steroids that do not aromatize heavily. If we do use a long acting ester, we would prefer to use one that doesn’t aromatize heavily, if the injectable does aromatize significantly, we would prefer to use a short acting ester as short acting esters don’t pool up, and an anti-aromatase would be a good idea.

Best fat burners: Clenbuterol and Cytomel

Best steroids for cutting: Testosterone Propionate, Equipose, Primobolan, Trenbolone, Winstrol, Proviron, Anavar

Post Cycle Therapy (PCT):
When you use any steroid, your HPTA will be suppressed. What this means is that your system is not producing any endogenous testosterone, which means you won’t have any hormone to help maintain your gains. What good is a cycle if you can’t keep your gains? So the key to cycling is to get your endogenous test back on track ASAP.

One thing that will hinder HPTA activation is excess estrogen, whether it is from aromatizable steroids used in your cycle or whether it be endogenous estrogen. Using anti- estrogens like Clomid and Nolva will help prevent this negative feedback.

When your body sends out LH (leutinizing hormone), it signals your testicles to begin producing test again. During your cycle, LH release will be suppressed and will remain suppressed for a few weeks after your cycle. HCG mimics LH and helps your testicles start producing testosterone. For our purposes we should view HCG as a “bridge”
between your cycle and the time your LH returns to normal function. However, HCG when used to heavily or for too long will actually suppress natural test production so it can be counter productive.

Different cycles will suppress your HPTA to different degrees. Cycles including Deca will be more suppressive than cycles including Var and Primo. Following is a post cycle therapy program that should help you recover from any sane and sensible cycle.

Before we outline the universal post-cycle therapy, we need to define when a cycle officially ends. If you are using long acting esters, your cycle ends 2-3 weeks after you take your last shot of the long ester. If you are using ONLY short acting steroids OR your last shot of long acting steroids was over 3 weeks ago, and the only thing you’ve been
running since then is short acting steroids, then your cycle officially ends the last day of administration of your steroids.

So given that, here is the universal post-cycle recovery program:

2 Weeks Before End of Cycle: HCG @ 1500IUs 3 times a week
1 Week Before End of Cycle: HCG @ 1500IUs 3 times a week
First Week Post-Cycle: HCG @ 1500IUs 2 times a week

Day 1 Post Cycle: Clomid @ 300mg
Days 2-14: Clomid @ 100mg ED
Days 15-28: Clomid @ 50mg ED

Days 1-28: Nolva @ 20mg ED

Excess Dietary Protein: Bodybuilding or Fat Storage??

Protein digestion begins in the stomach and ends in the small intestine

While the physical breakdown of proteins does take place in the mouth, it’s not until proteins reach the stomach that any appreciable chemical breakdown occurs; this is facilitated by hydrochloric acid (HCl) and the enzyme pepsin (converted from its inactive form, pepsinogen). Once the initial protein denaturing and peptide cleaving is complete, the product polypeptides pass through the pyloric sphincter of the stomach and into the proximal small intestine. The proximal small intestine (the duodenum to be exact) is where most of the digestion of proteins and virtually all of the absorption of amino acids occurs (a very small amount do get excreted in the feces). Here even more digestive enzymes are present to break down the remaining polypeptides into their individual amino acids along with some trace amounts of di- and tri-peptides. Once broken down completely, the free amino acids and di-/tri-peptides can then enter the cells of the small intestine where some (especially glutamine) are used for energy within the intestinal cells, with the remaining passing through into hepatic portal circulation. Those that do pass into circulation are destined for the liver.

Protein absorption claims

Before we head on over to the liver and discuss amino acid metabolism with regards to the initial claims, I would first like to touch upon another related (or variant) claim that some of you may have heard in the lay media or from an uneducated classmate, etc. It usually reads: “The average person can only absorb 30 grams of protein at one sitting. Anything above that will be stored as fat.” Unlike the claims in the introduction, this one offers no context whatsoever. Moreover, it’s downright moronic. While this assertion sounds like straw man argument, readily poised for the takedown, I actually got this gem of wisdom from an online article written by a Registered Dietitian. Believe me; I couldn’t make this crap up if I wanted to (note: this isn’t to slander the RD profession, this just an example which happened to involve someone who should know better).

For instance, let’s take someone who eats, dare I say it, 40 grams of protein in one “sitting” (whatever that means). If we are to assume only 30g are absorbed at a time, then it’s safe to say that the extra 10g will be excreted in the feces. This, however, is just plain false. The gut’s ability to absorb protein is somewhere in the ballpark of 95% (i.e. only ~2g aren’t absorbed in our example meal). Nevertheless, based on the initial argument, how are you supposed to store 10g of excess protein as body fat if you can’t even (allegedly) absorb it in the first place? Most people (who I can understand don’t have a degree in nutrition) don’t realize the difference between utilization and absorption and make this error. I’m not quite sure what this RD’s excuse is, because, in order to store or metabolize a nutrient (i.e. utilize it), it must first be absorbed into the body.

Your body will take its sweet time absorbing protein, no matter the amount. And this will vary from source to source. In the end, however, 30g is just an arbitrary number that has no roots in scientific evidence. At this point let us circle back to the initial claim, that excess protein, which has already been absorbed, during times of adequate energy and carbohydrate intake, is predominantly converted to fatty acids and stored as body fat.

Liver, the primary site for amino acid metabolism

As we’ve already covered, the amino acids released from the small intestine are destined for the liver. Over half of all the amino acids ingested (in the form of protein) are bound for and taken up by the liver. The liver acts almost as a monitor for absorbed amino acids and adjusts their metabolism (breakdown, synthesis, catabolism, anabolism etc.) according to the body’s metabolic state and needs. It is here the initial claim comes into play. While the pathways for fatty acid synthesis from amino acids do exist, no argument there, the statement that all excess protein, under specified conditions, will be stored as fat ignores recent evidence.

Building Muscle and Losing Body Fat: Plateau Busting Rules

Building muscle while simultaneously losing body fat is a delicate balance.. We stop making progress and think we’ve hit a plateau. This article will address both small errors that require a simple change, and more complex solutions from our bag of tricks for busting any fat loss-muscle building plateau. We’ll start with simple answers and finish with more complex solutions.

Use Correct Training Technique

Unless you’ve been taught how to train by an experienced coach, your training technique is probably lacking and it’s the reason you’re not making progress. Bad technique can mean a lot of different things:

  • You could be haphazardly raising and lowering the weights without any awareness of the speed of the muscle contraction.
  • You could have wacky training posture that leads you to use muscles you’re not intending to train.
  • You could be doing partial range-of-motion exercises when you need to do full range training at this stage.
  • You could be using too light a weight to produce any change in your body—ladies, this means you.

Plan Your Workouts in 2 to 4 Week Phases

Avoid boredom and stagnation by planning your workouts in 2 to 4 week phases. This is a very basic principle of strength training because the body responds rapidly to the stress of training loads.
Solve It: There are two basic parameters for planning your set/rep schemes for fast results:

  1. First is accumulation, which requires doing more reps and sets so that you spend more “time” under the weight. The primary change to your body with accumulation training is more muscle and less fat.
  2. Second is intensification, which refers to lifting heavier weights that are closer to the maximum amount you can lift so you get stronger. The primary change to your body is greater strength. 

Even if your only interest is a killer physique and you can’t care less about strength, you need to alternate between these phases in order to avoid the dreaded plateau. You’ll get better body comp results in the end because the intensification phase will allow you to use heavier weights once your back to pushing volume.

Get Strong In the Big Lifts Before Doing Isolation Training

Isolation and other “plateau busting” lifts can help you make progress once you’ve put in the sweat and tears and achieved a threshold of strength in the big lifts like deadlifts, squats, and presses.

We’re talking a strength threshold of at least 1.5 times body weight for full squats and about the same for bench press, although the bench press number may be lower for women.

Solve It: First, focus on free weight multi-joint lifts and avoid machines or isolation training.
Second, use proper form, and plan your training using the parameters listed in 2
Third, track your progress by writing it down. This will allow you to be honest about where you are and get tiny victories, like an extra rep or an extra kilo, which can sustain motivation during the hard times.

Do Short Strongman or Sprint Workouts To Promote Fat Loss

Prioritize cardio and you will never be satisfied with your physique. If you’re doing regular cardio, it could be a primary cause of your lack of progress. Instead, do brief strongman or sprint workouts for optimal leanness.
It’s a well-known fact in sports science that aerobic-type cardio “turns off” muscle building pathways, inhibiting muscle growth. It’s also possible that when you pair weight workouts and cardio in the desperate hope that you’ll lose body fat, the high volume of exercise elevates cortisol, further impairing muscle growth.
Solve It: Ditch anything that resembles steady-state cardio in favor of interval training or strongman workouts. Workouts should be less than 25 minutes long and metabolically taxing with short rest periods and intense work bouts.

Vary Your Lifting Speed & Always Count Tempo 

Tempo refers to the speed with which you perform the up and down phases of any lift. A general principal for improving your body comp is to favor moderate eccentric (3 to 6 seconds) and fast concentric tempos for a longer time under tension. This is because slow-speed lifting bring about more metabolic adaptations, such as increases in muscle glycogen, creatine phosphate, and ATP, which is what you want when trying to change your body. In contrast, explosive ballistic contractions such as Olympic lifts bring about more central nervous system adaptations. Yet, if you’re a savvy trainee, you’ve probably learned that always training for a metabolic effect will lead to stagnation, so periodization is key.
Solve It: A combination of high- and low-velocity training produces greater strength and body composition results than either one alone. Try a fast tempo for both the eccentric and concentric phase of the lift to maximize the peak loads experienced by the body.
Lowering the weight fast coupled with an explosive upward motion activates gene signaling and produces greater protein synthesis, while recruiting distinct type II muscle fibers that aren’t used during slower movements.

Use Partial Range-of-Motion Training

Full range training, such as deep squats or deadlifts on a platform, is a basic principle of proper training for strength and body composition. But, training partials in which you break down a rep into individual parts is an incredible tool for overcoming a plateau. Often, plateaus are about sticking points, which is the part of a lift where you are weakest. For example, the sticking point in the deep squat or bench press is typically right around midpoint, although, the entire bottom segment (bottom to parallel in the squat) can give lifters trouble.
Solve It: When performing partial-range-of-motion training, always incorporate full-range-of-motion sets within the workout. Many people make the mistake of doing just partials, but research shows this leads to diminished returns and less muscle development.

How To Use Deadlifts To Reach Your Genetic Potential

Stalled progress in the deadlift is maddening, especially after you’ve made rapid gains as a novice. Here is a program from PICP coach Jeff Banda that will help you bust that plateau:

The Program

A1) Top 1/4 deadlifts in rack, 4 x 4-6 reps, 2210, rest 100 seconds
A2) Lying leg curls, feet neutral, 4 x 4-6 reps, 4010, rest 100 seconds
B1) Top half deadlifts in rack, 4 x 4-6 reps, 2210 tempo, rest 100 seconds
B2) Lying leg curls, feet outward, 4 x 4-6 reps, 4010 tempo, rest 100 seconds
C1) Full-range deadlift, 3 x 6-8 reps, 4010 tempo, rest 90 seconds
C2) Lying leg curl, 3 x 6-8 reps, 4010 tempo, rest 90 seconds

Here’s why it works: Top ¼ deadlifts are where you’re strongest and they offer the most advantageous leverage. Use a super heavy weight, which you could never even think of getting off the ground at this point, to recruit the highest threshold motor units in the muscle.
It also desensitizes components of the tendon that inhibit strength (your body’s built-in attempt to protect you from yourself), giving you both a physical and mental advantage. Top half deads overload a common trouble spot, and since you’re performing them after the ¼ deadlifts, you’ll get the benefit of having activated muscle fibers with the super heavy load. Full range deads overload the most common sticking point, which is the pull off the ground.

Use Isometrics To Target Weak Links

Isometrics are unique for their ability to produce high levels of muscle tension without a change in the muscle’s length or joint angle. You’re probably familiar with the classic but not very useful isometric, the plank.
Using isometrics within a dynamic or “moving” contraction increases neural drive to maximize the strength potential of newly developed muscle.
Solve It: One-and-a-quarter contractions are a highly efficient strategy for overcoming plateaus. The most well known version is one-and-a-quarter squats in which you go all the way down, come up 20 to 30 degrees, pause for a second, descend back to the bottom and come up quickly. The one-and-a-quarter method can be used for other lifts such as leg curls, bench press, or even bicep curls. More advanced trainees should try two to three pauses during the concentric contraction so as to hit all parts of the muscle for growth and strength.

Use Chains

Training with chains is a surefire way to bust plateaus because it matches the resistance curve to the human force curve. For example, if you put chains on the ends of the barbell when squatting or bench pressing, the chains will pile up on the floor during the down portion of the lift, decreasing the weight. As you come up out of the squat, the weight will increase as the chains come off the floor and contribute to your load. Your load gets heavier at the point where you are weakest, requiring you to train through the sticking point of the lift. In one study of D1 college football players, those who trained with chains increased maximal strength by 42 percent more than those who trained with traditional lifts. Researchers note that weighted chains supply the added benefit of training the stabilizer muscles because they swing and oscillate throughout the range-of-motion of a lift.
Use It: Chain training can be used when training the extensor muscles in lifts like the bench press or squat. Research suggests the use chains weighing 10 to 15 percent of the barbell weight, rather than a lighter 5 to 8 percent. So, if the loaded barbell weight is 100 kg, each chain should weigh 10 to 15 kg.

Do Complex Training With Fast Eccentrics

Have you heard of the stretch-shortening cycle? It’s the elastic component of the muscle and tendon, which makes you springy.
Research shows that being strong is not enough. To achieve your athletic potential, you need to actually train the SSC.
Solve It: An example of complex training is to do a heavy strength exercise followed immediately by a fast power exercise, such as squats at 95 percent of the 1RM followed by vertical jumps. For upper body, doing a heavy bench press followed by a bench throw is standard, but you could also try doing the heavy lift prior to a combat sports that require peak upper body.

Use Nutrition To Support the Immune System

Nutrition will make the most difference if you’re nearing your body’s physical limit from training. If all you need to do is work harder, the benefit of nutrition is negligible. Here Are A Few Tricks: Increase your fat intake. Try butter (it supplies butyric acid, which feeds beneficial but bacteria), coconut oil (it provides a quick fuel source and is anti-inflammatory), and fish (omega-3 fats that are anti-inflammatory).
Eat probiotic foods to give your gut some love. Try sauerkraut, pickles, kim chi, pickled ginger, high-quality yogurt, and the beverage kefir.
Bump up calories once a week as high as 150 percent. Adequate calories are essential for recovery and eating enough tells the body that resources abundant, making it more responsive to pack on muscle and give up body fat. It’s the opposite effect of “starvation mode” in which calories are restricted and your body ratchets down your metabolic rate.
If your plateau is more diet and fat-loss related, check out this article for specific tools to help you take it to the next level.

GHRP-6 (Growth Hormone Releasing Hexapeptide)

GHRP-6 is an injectable peptide in the category of growth hormone releasing peptides, or GHRP’s. The most common use of these peptides is to increase growth hormone production. With regard to increasing growth hormone, all of these work similarly, and there is no need or advantage to combining them. Instead, the one most suited for the particular case is chosen.

The principal use of GHRP-6 is to provide increased growth hormone levels, which also results in increased IGF-1 levels. This aids fat loss and in some instances aids muscle gain as well. Generally, GHRP use is chosen as an alternate to GH use, and only rarely is combined with growth hormone. Reasons to prefer GHRP-6 over other GHRP’s are its effect on increasing appetite (which also can be a disadvantage), and its demonstrated value in reducing inflammation and helping to heal injury, particularly tendinitis.

When to use GHRP-6

GHRP-6 is most generally used for the same purposes that growth hormone might be used, but may be chosen where a cost advantage exists favoring GHRP-6, growth hormone is not available, or the individual prefers the idea of stimulating his own growth hormone production to injecting growth hormone. These purposes can include increased fat loss, improved muscle gain when used in combination with anabolic steroids, cosmetic improvement of the skin, and improved healing from injury.

There is no need to cycle GHRP use, and so therefore GHRP-6 may be used whenever it is thought to be useful for any of the above reasons. However, when usage is ongoing instead of short-term amplification of a steroid cycle or fat-loss program, I recommend limiting dosage to the minimum end of the suggested dosage range.
Increased effect from combining GHRP-6 with a GHRH

While GHRP-6 is capable of inducing large increases in growth hormone production when used alone, a given dose will show markedly more effect what a GHRH (growth hormone releasing hormone) peptide is taken at the same time. Alternately, when combining a GHRH with GHRP-6, only about half or a third as much GHRP-6 is needed to obtain the same increase in growth hormone production. For this reason, it’s a common choice to combine a GHRH with a GHRP, but excellent results are often obtained without doing so. A preferred choice of GHRH is Mod GRF 1-29 (also known as CJC-1295 without DAC.)

How to use GHRP-6

GHRP-6 is most commonly provided in small vials of 5 mg, which should be stored under refrigeration. (It is acceptable however for them to be mailed unrefrigerated.) The vial is diluted with a convenient volume of sterile or bacteriostatic water. For example, the vial might be diluted with 2.5 mL of water, yielding a solution of 2 mg/mL (2000 mcg/mL.) After the water addition, the vial again will be stored under refrigeration.

When dosing, an appropriate volume will be drawn from the vial with (typically) an insulin syringe, according to the desired dose and the concentration of the preparation. In the above example, a 100 mcg dose would require only 0.05 mL, or “5 IU” as marked on an insulin syringe. A 300 mcg would require 0.15 mL, or “15 IU” as marked on an insulin syringe.
Injection may be subcutaneous, intramuscular, or intravenous according to personal preference.

Dosing will ordinarily be at least twice per day and preferably 3x/day for best effect, taken at least 30-60 minutes before a meal and at a time of non-elevated blood sugar (in other words, after blood sugar has had time to fall since the most recent meal.) The amount taken generally will be from 50-300 mcg at a time. When using a GHRH along with GHRP-6, dosing should be reduced to 50-100 mcg at a time.

For increase in GH levels, higher doses within the suggested range definitely increase effect. With regard to healing benefit, for example for tendonitis, the low end of the range is often entirely sufficient and noticeably greater effect is not necessarily seen with increased dose.

While there is no sharp cut-off between a solution of GHRP-6 still being good and having lost potency with time, as a general guideline, a vial should be used within a month of having been reconstituted. Past this, I would discard the vial and start a new one.

Pharmacological class: Mimetic of ghrelin

GHRP-6 and all GHRP’s are mimetics of ghrelin, a hormone produced by cells of the stomach in response to a fasted condition, including brief fasts. Ghrelin and ghrelin mimetics work by activating the ghrelin receptor, also called the growth hormone secretagogue receptor (GHS-R1a). Elevated ghrelin levels act towards increasing growth hormone levels by stimulation of ghrelin receptors in the pituitary.

Injection of GHRP-6 or any GHRP stimulates growth hormone release in essentially the same way as fasting-induced elevation of ghrelin levels. The effect can be markedly greater, however. Supraphysiological levels of growth hormone are easily achieved with proper dosing of any GHRP.

Other activities of ghrelin, and potentially of GHRP-6

Ghrelin has many activities in the body besides stimulating growth hormone release. It stimulates appetite, is cardioprotective, can help protect cells against oxidative damage, can reduce inflammation and promote healing, and can promote fat-burning in muscle. There is also some effect on increase in cortisol production via increase in ACTH, and increase in prolactin. However, where the activity of ghrelin is comparable to that which ordinarily occurs during fasting, effects on cortisol and prolactin likewise are comparably only to that experienced while fasting.

Ghrelin itself so far as I know has not been used as a performance-enhancing drug. In contrast, experience with GHRP’s is quite extensive. GHRP-6 in particular has in many cases been effective in treating tendonitis, which may well be due to its activity at the ghrelin receptor.

Broadly speaking, it’s long been a widespread view that fasting can in many instances provide healthful effects beyond simple fat loss. It’s speculative to say that increased ghrelin levels must be a major cause of such effect (if granting the effect), but it’s entirely consistent with the scientific literature that such elevation of ghrelin levels may have health benefits. Appropriate-dosed and cycled GHRP use may at least partially provide such benefits, particularly with regard to anti-inflammatory and healing effect.

There are indications in the literature of ghrelin acting to promote fat-storage, but this appears to be tissue-specific, for example in the liver. In bodybuilding, by no means has any such effect been apparent with GHRP-6. Instead, GHRP-6 has been effective as a cutting drug, provided that food intake is controlled.

Because some GHRP’s are equally effective as others in increasing GH but differ in effect on hunger or ACTH stimulation, it seems likely that there may be differences in ghrelin receptors between different tissues, or differences in function (for example with cofactors.) This is the most likely explanation for GHRP-6 being effective in stimulating hunger and helping heal tendinitis, while GHRP-2 stimulates hunger less and may have less value for healing.

Factors decreasing efficacy of GHRP-6

For increasing GH levels, GHRP-6 is less effective in the presence of high blood glucose levels or high somatostatin levels, which result from high IGF-1 levels. For this reason, for best effect GHRP-6 should be taken while blood sugar is relatively low, for example about 30-60 minutes before a meal. GHRP-6 will have reduced effect if growth hormone is being taken by injection, because growth hormone increases IGF-1. Where growth hormone use is limited to no more than about 14 IU per week, simultaneous GHRP-6 use probably still will increase GH somewhat further, but if growth hormone use is greater than this then likely GHRP-6 injections will do little to nothing towards increasing growth hormone levels any further.

Low thyroid levels also decrease efficacy of GHRP-6, and modestly increased thyroid levels, such as with 50-200 mcg/day of T4, or 12.5-50 mcg/day of T3, may improve efficacy. (If T3 is used, dosage should be divided into three doses per day.)

GHRP-6 and gynecomastia

While I doubt that GHRP-6 use is likely to cause gynecomastia (gyno), elevation of prolactin from GHRP-6 use can be problematic for some individuals who already have gyno, including undetected gyno which developed during puberty. Ipamorelin can be a better choice of GHRP for such individuals.

GHRP-6 can effectively provide substantial increases in GH production. It’s typically taken 2-3 times per day by injection at times when blood sugar is not elevated. Cost is generally moderate. The only common potential adverse side effect is increased hunger. Common alternates include GHRP-2, hexarelin, ipamorelin, or growth hormone itself. GHRP-6 also may provide benefits which GH does not, via its action at the ghrelin receptor in various tissues of the body.

Boldenone by Jintani Labs - It’s true potential

Boldenone, particularly Boldenone Undecyclenate, is a drug which has procured devoted advocates on both sides of the fence. Opinions run strong regarding the effectiveness of this steroid, with the unimpressed often chastising it as worthless and its ardent supporters touting it as indispensible. Perhaps more than any other steroid, there is a clear rift between acceptance and rejection and while variances in personal response combined with its slow-acting nature no doubt play a role in this divide, I am convinced that the primary reason for such conflicting views is due to suboptimal dosing practices among many of its users.

Boldenone, most commonly known as Equipoise was originally produced as a veterinary steroid. Traditionally, it has been used in race horses in order to increase performance and aid in recovery, but it did not take long for bodybuilders to begin experimenting with the drug and by the 1980’s, it was in relatively high demand. Initially thought of as an “exotic” steroid, it is now under mass production by today’s UGL’s and is considered basic stock in the world of anabolic steroids. Legend has it that even 8X Mr. Olympia himself, Lee Haney, was a big fan of this drug. Certainly, this would’ve contributed to its growing popularity back in the day.

On paper, there doesn’t appear to be anything special about Boldenone. Here are some basic facts: It is a non-progestagenic, non-methylated 1-dehydro derivative of testosterone, so it lacks the liver toxic effects associated with most oral steroids. It possesses an A:A ratio of 100:50 (anabolic/androgenic), making it roughly half as androgenic as testosterone and equally anabolic when compared on a mg to mg basis. Boldenone displays a weaker binding affinity for the androgen receptor than testosterone, yet it is less prone to attach to sex hormone binding globulin, which likely helps to balance out its myotropic effects. The drug will maintain blood levels of the base compound for roughly 14-16 days post-injection, yet twice weekly injections are appropriate when attempting to maintain stable blood levels.

One interesting characteristic of Boldenone which bears mentioning is the discrepancy between its proposed rate of aromatization and its propensity to initiate estrogen side effects. Boldenone is said to aromatize at about 50% the rate of testosterone, which should make it a fairly estrogenic drug, prone to causing estrogenic side effects such as gynecomastia and water retention, especially at higher dosages. However, real-world experience has shown us that Equipoise is very unlikely to propagate serious estrogenic side effects, even when administered at high dosages. For example, it is very common for a testosterone user to experience gynecomastia and water retention at a dosage of 500 mg per week, if not dealt with…and they almost always occur when venturing up to the 750-1,000 mg per week range. Many users will encounter these side effects even when using as little as 300-400 mg per week. However, when administering Equipoise at dosages of 600-800 mg per week, those side effects rarely manifest…and few users experience them even when using 1,000 mg per week or more! With Equipoise aromatizing at half the rate of testosterone, one would expect to witness estrogenic side effects in a large percentage of users, but this does not occur. Why? One explanation is found in the drug’s metabolism. Specifically, Boldenone metabolizes to an anti-aromatase inhibitor known as 1,4 dienedone. This could certainly explain why Boldenone, despite it relatively high rate of aromatization, does not deliver anywhere near the estrogenic punch that its aromatization rate implies.

One highly prized trait of Equipoise, which has been established as a long-held belief for decades now, is its ability to increase vascularity to a greater degree than other androgens. Many years ago, I couldn’t help but wonder if this claim was nothing more than placebo-strengthened lore. Yet, repeated personal experiences, as well as speaking with 100’s of other athletes over the years, have confirmed, at least in my mind, this claim to be fact. It is true that all androgens increase RBC count, which can potentially increase vascularity, yet science has not yet found any mechanism by which Boldenone might accomplish this more proficiently than other anabolic steroids. Still, I do not find this surprising given the fact that we know many things about anabolic steroids through real-world experience, which have not yet been proven by accepted science.

In light of the vascularity claim, this is an ideal time to discuss another one of Boldenone’s unique, yet scientifically unproven characteristics…increased appetite. While once again, we can find no legitimate mechanism by which Boldenone might accomplish this feat, many users have sworn that this “side effect” is a reality. Personally, I am not yet convinced of its existence, as there are not only other plausible explanations, but I have never experienced it myself, despite experimenting with large dosages of the drug. Still, I do not deny the possibility of its existence, being that many individuals over the years have supported the notion.

As expected, Boldenone is known to provide lean, hard, water-free increases in muscle tissue. However, it is not generally recognized as a very potent muscle-builder, but has often been used as an “add-on” or secondary steroid, in order to further increase one’s results without a commensurate increase in side effects. Few expect it to provide impressive growth and it is almost never used as the base drug in one’s stack. Because of this, I began to re-evaluate the proposed optimal dosing guidelines for this drug and what I found that is that unlike most other steroids, which usually begin to reach the point of diminishing returns at between 500-1,000 mg per week, Equipoise tends to reach its optimal dosing range much later on…around 2 grams per week (great results will be found between 1.5-3.0 grams). Of course, all steroids will continue to activate more receptor sites (and thus provide greater muscle growth), up until about 3,500 mg per week, but with Boldenone, it seems that the gains are just starting to get good at 1 gram weekly, while most other AAS seem to be nearly tapped out by that point, quickly hitting the point of diminishing returns.

One factor which makes higher dosing with Equipoise possible is its relatively mild nature, in terms of side effects. While many steroids would be rather unpleasant to run at 2 grams weekly, many individuals will find Boldenone to be rather tame at that dose. This is an important consideration because after all, no one wants to be miserable on-cycle. It is understandable to think that some may initially be put off by these doses, as the typical steroid user frequently views anything over 1 gram per week (per steroid) as “mega-dosing” and therefore, excessive. Assumably, this mentality was instilled through the repeated dosing recommendations of other anabolic steroids, which by their nature, do not need to be dosed much higher than 1 gram weekly in order to achieve near optimal results, but with Boldenone, we are dealing with a different animal altogether. Boldenone is a steroid which “needs” to be dosed higher than other anabolic steroids if one wishes to take advantage of the full capabilities of this drug. 600-800 mg per week just isn’t going to cut it if one wants to experience everything this drug has to offer. When dosed at the above mentioned range, Boldenone becomes a formidable muscle-builder, packing on genuine muscle fiber rather quickly. No, using bold at 2 grams weekly will not add as much bodyweight as 2 grams of test, but unlike testosterone, the weight gained will be actual muscle tissue, not a moderate amount of muscle accompanied by a small pool of water. In short, you will look much better using 2 grams of Boldenone along with a moderate amount of test, than you will be using a mountain of test along with a moderate dose of Boldenone.

Over the last 6+ months, at my recommendation, I have had the pleasure of witnessing first-hand the effect that larger dosages of Boldenone has had on numerous individuals and without exception, everyone who engaged in this practice reported a much more pleasant cycle compared to the typical test-heavy cycles so common today, as well as a dramatic improvement in the quality of their musculature. Needless to say, by the conclusion of their cycles, Boldenone was no longer viewed a weak muscle builder, being suitable only as an “add-on”, but as a real alternative to more traditional cycles. So, the next time you consider using Bold as a part of your cycle, try to look at it from a fresh perspective…as a drug which “needs” to be utilized at higher dosages in order to experience all that it has to offer. If you do, I can promise you impressive, high-quality, water-free increases in muscle tissue, along with dramatic increases in vascularity over a 12-16 week period.

HGH Usage and Dosage for Women

Women need to take comparatively higher doses of HGH than men, to experience the wonderful results of the replacement therapy. Nevertheless, low doses of the hormone will not cause any harm, while high doses for prolonged periods of time, may lead to breast cancer and other complications in women.

Expected good results were observed in some women who were on 2 IU /day dosage for six months. The results demonstrated a decrease in the waist diameter by about four inches. Some women who have used the HGH therapy, have an opinion that based on the goals, the dosages must be determined. They have come to the conclusion that six to twelve IU /day, in combination with anabolic androgen steroids and insulin would enhance muscle growth tremendously. While the dosage for fat reduction and enhancing the lean muscle mass was found to be three to six IU/day.

High doses of the growth hormone in women also causes fluid retention. Also, in some women, it was observed that high dosages of the hormone increases the blood pressure in the body. This may not have grave repercussions in all women, but it definitely can create problems in those who have hypertension. So, it is advisable for women who are suffering from high blood pressure, to begin the replacement therapy with an initial HGH dose of around 0.5 IU/day and gradually step it up.

It has been discerned from innumerable case studies, that estrogen affects the action of HGH in women. Probably, that could be the reason why women cease to grow tall, before men do. Also, women experience fewer results for the same dosage of the hormone, that is used in men. Optimal dosage administration in women must consider the 28-days cycle. Take the average of that dose by dividing it by 28. Let us assume that the dose is 1 IU/day. This dose must be used for 1 to 12 days of the cycle. Then, half of this dose, i.e. 0.5 IU/day, must be used from the 13th day till the 16th day of the cycle. Then, the dosage of 1.5 IU / day must be used from the 17th day till the 28th day of the cycle. By doing this, you can maintain an average dose of 1 IU/day, all through the cycle. The changes in the dosage would interact with the levels of estrogen and progesterone effectively, and yield the best results.